The ALS Association - Knock ALS Out of the Park
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Selective neuronal degeneration in MATR3 S85C knock-in mouse model of early-stage ALS
Genetics of amyotrophic lateral sclerosis: seeking therapeutic targets in the era of gene therapy
Postmortem Cortex Samples Identify Distinct Molecular Subtypes of ALS: Retrotransposon Activation, Oxidative Stress, and Activated Glia - ScienceDirect
Targeting mitochondrial Ca2+ uptake for the treatment of amyotrophic lateral sclerosis - Zhong - The Journal of Physiology - Wiley Online Library
Genes, Free Full-Text
The ALS Association - Knock ALS Out of the Park
Disrupted autophagy and neuronal dysfunction in C. elegans knockin models of FUS amyotrophic lateral sclerosis - ScienceDirect
Biomolecules, Free Full-Text
ALS Association: Knock ALS Out of The Park • Ads of the World™
Clinical-Genetic Features Influencing Disability in Spastic Paraplegia Type 4
ALS-linked FUS exerts a gain of toxic function involving aberrant p38 MAPK activation
A blood–brain penetrant RNA-targeted small molecule triggers elimination of r(G4C2)exp in c9ALS/FTD via the nuclear RNA exosome
Prospects for gene replacement therapies in amyotrophic lateral sclerosis
ALS Association: Knock ALS Out of The Park • Ads of the World™
Human iPSC-derived astrocytes from ALS patients with mutated C9ORF72 show increased oxidative stress and neurotoxicity - eBioMedicine
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